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我深愛的雷奈、費里尼及其他

為了解決大 師兄 電影 完整版的問題，作者李幼鸚鵡鵪鶉,鄭秉泓 這樣論述：

　　從　　雷奈《穆里愛》與費里尼《愛情神話》　　到　　楊德昌《牯嶺街少年殺人事件》是托爾斯泰文學∕奧黛麗．赫本電影的一九六○年代台灣社會背景版的《戰爭與和平》　　到　　我跟蔡明亮都愛小劇場∕電影∕李香蘭的夢般喜悅 　　二十年來的重量級評論重新編輯整理　　文字天馬行空、喃喃自語又旁徵博引　　從歐美到亞洲，李幼鸚鵡鵪鶉最完全、最私密的電影觀點 　　若你每年都到影展朝聖，必定見過一位身形瘦削，頂著一頭瘋亂長髮，總是拿著照相機四處拍攝他眼中「美的事物」的奇人。他從前叫做李幼新，如今叫做李幼鸚鵡鵪鶉，不過前陣子他終於把留了多年的長髮剪掉，不變的是，他依舊從大師經典到好萊塢強片再到學生畢業製作持續尋尋

覓覓，在電光幻影中尋找他自己。 　　李幼鸚鵡鵪鶉見證了台灣小劇場的起與落，參與了台灣電影不同時期一場接著一場的美好戰役，憑藉他驚人的記憶力與獨到的切入方式，為台灣影史留下一篇又一篇的精彩紀錄。他癡迷奧黛麗．赫本、深愛雷奈與費里尼，執著於電影中的「看」與「被看」、「同」與「異」，致力在不同影像文本中挖掘各式各樣的「2」（二元對比）。 　　《我深愛的雷奈費里尼與其它》是李幼鸚鵡鵪鶉自從1993年的《關於雷奈∕費里尼∕電影的二三事》、《男同性戀電影…》之後的首本影評集（這期間僅有自傳《鵪鶉在鸚鵡頭上唱歌》問世），不僅有將近二十年來他在各大報章雜誌發表過的精華蒐錄，還有多篇私房手稿首度曝光，總計「奧黛

麗．赫本與『泛奧黛麗．赫本電影』」、「從雷奈與費里尼開始」、「用電影解釋電影 關於雷奈與費里尼的影響」、「關於楊德昌」、「只有蔡明亮」、「我 私人的 台灣電影」、「還有華語電影」、「現在是二十一世紀」、「以布萊德．彼特結束」九大單元，超過一百二十篇精彩文章、多達二十七萬字，如他所言，這其實是李幼鸚鵡鵪鶉版本的追憶似水年華。 　　不僅寫給他自己，也寫給全天下影癡…… 本書特色 　　1. 李幼鸚鵡鵪鶉二十年來第一本影評集　　2. 包括多篇未曝光的私房手稿　　3. 影評之外，亦可一窺李幼鸚鵡鵪鶉的影痴生涯與生活理念 作者簡介李幼鸚鵡鵪鶉 　　每週在《破》周報專欄發表影評，深愛奧黛麗．赫本、雷奈和費

里尼。於1985年率先編寫《港台六大導演》與《電影、電影人、電影刊物》兩本「台灣新電影」論述；於1989年率先為金馬國際影展規劃「台語片整理與回顧」專題，著有《坎城－威尼斯影展》、《男同性戀電影…》、《鵪鶉在鸚鵡頭上唱歌》等書。拍攝過短片《生活像電影──關於奧黛麗．赫本的二三事》與《鵪鶉在鸚鵡頭上唱歌》。並在蔡明亮、陳俊志、黃銘正、吳米森、傅天余的電影與鴻鴻的舞台劇參加演出。2011年獲頒台北電影節卓越貢獻獎。 編者簡介 鄭秉泓 　　高雄人，以英文名字Ryan在「關於電影，我略知一二」個人部落格及各類電影刊物上發表影評，國藝會首屆「台灣藝文評論徵選專案」視聽媒體藝術類首獎兼特別獎得主，著有《台

灣電影愛與死》。

大 師兄 電影 完整版進入發燒排行的影片

An Integrated Approach For Uncovering Novel DNA Methylation Biomarkers For Non-small Cell Lung Carcinoma

為了解決大 師兄 電影 完整版的問題，作者VAIBHAV KUMAR SUNKARIA 這樣論述：

Introduction - Lung cancer is one of primal and ubiquitous cause of cancer related fatalities in the world. Leading cause of these fatalities is non-small cell lung cancer (NSCLC) with a proportion of 85%. The major subtypes of NSCLC are Lung Adenocarcinoma (LUAD) and Lung Small Cell Carcinoma (LUS

C). Early-stage surgical detection and removal of tumor offers a favorable prognosis and better survival rates. However, a major portion of 75% subjects have stage III/IV at the time of diagnosis and despite advanced major developments in oncology survival rates remain poor. Carcinogens produce wide

spread DNA methylation changes within cells. These changes are characterized by globally hyper or hypo methylated regions around CpG islands, many of these changes occur early in tumorigenesis and are highly prevalent across a tumor type.Structure - This research work took advantage of publicly avai

lable methylation profiling resources and relevant comorbidities for lung cancer patients extracted from meta-analysis of scientific review and journal available at PubMed and CNKI search which were combined systematically to explore effective DNA methylation markers for NSCLC. We also tried to iden

tify common CpG loci between Caucasian, Black and Asian racial groups for identifying ubiquitous candidate genes thoroughly. Statistical analysis and GO ontology were also conducted to explore associated novel biomarkers. These novel findings could facilitate design of accurate diagnostic panel for

practical clinical relevance.Methodology - DNA methylation profiles were extracted from TCGA for 418 LUAD and 370 LUSC tissue samples from patients compared with 32 and 42 non-malignant ones respectively. Standard pipeline was conducted to discover significant differentially methylated sites as prim

ary biomarkers. Secondary biomarkers were extracted by incorporating genes associated with comorbidities from meta-analysis of research articles. Concordant candidates were utilized for NSCLC relevant biomarker candidates. Gene ontology annotations were used to calculate gene-pair distance matrix fo

r all candidate biomarkers. Clustering algorithms were utilized to categorize candidate genes into different functional groups using the gene distance matrix. There were 35 CpG loci identified by comparing TCGA training cohort with GEO testing cohort from these functional groups, and 4 gene-based pa

nel was devised after finding highly discriminatory diagnostic panel through combinatorial validation of each functional cluster.Results – To evaluate the gene panel for NSCLC, the methylation levels of SCT(Secritin), FOXD3(Forkhead Box D3), TRIM58(Tripartite Motif Containing 58) and TAC1(Tachikinin

1) were tested. Individually each gene showed significant methylation difference between LUAD and LUSC training cohort. Combined 4-gene panel AUC, sensitivity/specificity were evaluated with 0.9596, 90.43%/100% in LUAD; 0.949, 86.95%/98.21% in LUSC TCGA training cohort; 0.94, 85.92%/97.37 in GEO 66

836; 0.91,89.17%/100% in GEO 83842 smokers; 0.948, 91.67%/100% in GEO83842 non-smokers independent testing cohort. Our study validates SCT, FOXD3, TRIM58 and TAC1 based gene panel has great potential in early recognition of NSCLC undetermined lung nodules. The findings can yield universally accurate

and robust markers facilitating early diagnosis and rapid severity examination.